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The treatment of tumors formed by the radio-resistant GSCs with an IGF1R inhibitor results in a marked increase in radio sensitivity, suggesting that the blockade of IGF1R signaling is an effective strategy to reverse radio-resistance (Osuka et al., 2013)
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13 , 16 Patients with moderate and severe HS can also develop folic acid deficiency resulting from increased folate utilization to support the chronic erythroid hyperplasia in the bone marrow
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