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This Review summarizes current knowledge of the biology of prolactin and its receptor, as well as its physiological and pathological roles
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Post-mortem analysis of human brains from AD patients showed that that Aβ 40 discriminated between AD patients and high pathology controls more readily than Aβ 42 (Gao et al., 40 oligomers have been shown to induce neurotoxicity and disrupt brain lipid bilayers while other amyloid species do not (Bode et al., 40 is induced by an increase in intracellular Ca 2+ , and this has led to the hypothesis that the accumulation of intracellular Aβ may occur as an early event in the pathogenesis of AD (Itkin et al., 2+ ] i and may enhance Ca 2+ -mediated excitotoxicity, which may induce progressive memory loss and cognitive deficits and enhance neuronal cell apoptosis
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