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A combination of an AAV-delivered angiogenic molecule such as vascular endothelial growth factor, bone morphogenetic protein signaling receptor (caALK2) and receptor activator of nuclear factor-kappa B ligand (RANKL) were demonstrated to promote bone allograft turnover and osteogenesis as a mode to enrich human bone allografts (60)
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Regarding the arsenic efflux, it has been reported that both MRP1 and 2 along with the phase II conjugating enzymes like glutathione transferase (GST) are the major contributors for the resistance towards specific metals and toxicants [71, 76]
It functions primarily to support fat utilization