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One notable mechanism involves the upregulation and/or activation of receptor tyrosine kinases (RTKs), leading to the phosphorylation of glucose transporters and their subsequent relocation to the plasma membrane (Schafer et al
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ME1 and ME3 require NADP + and ME2 utilizes either NAD + or NADP + for their catalytic activities, thus NADPH can be produced by ME both directly and indirectly through the activity of the NNT that catalyzes the transfer of hydride ions from NADH to NADP + and produces NADPH in mitochondria
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