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The Z polymorphism has shown an association with demyelinating and hypomyelinating conditions, potentially explaining the higher frequency of demyelinating diseases in individuals with A1AT pathological polymorphisms ( Furthermore, A1AT has been implicated in lipid metabolism
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Pickart and Margolina reported that GHK-Cu modulates expression of over 4,000 human genes in bioinformatic analyses, including genes involved in collagen synthesis, anti-inflammatory pathways, and DNA repair
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