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This results in lipid peroxidation, oxidative damage to membranes and other lipid-containing molecules, and ultimately to cellular damage and ferroptotic cell death ( Glutathione Metabolism On the other hand, inhibition of glutamate/cystine antiporter (system x c – , xc-, with xCT as the functional subunit of system x c – ) and depletion of GSH cause inactivation of GPx4, the critical antioxidant enzyme and regulator of ferroptosis (Seibt et al., 2019)

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All mAbs were purified in-house as described (61)