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Initially, excess copper binds to mitochondrial lipoic acid-related protein components (e.g., DLAT) (Rowland et al., 2018), leading to the formation of insoluble DLAT aggregates in mitochondria and the loss of FDX1-dependent Fe-S clusters
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*Correspondence: Chun Fang, [email protected] †These authors have contributed equally to this work This article was submitted to Food Microbiology, a section of the journal Frontiers in Microbiology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers
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