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The DAMP released by mitochondria (TLR, TNF receptor, inflammasome) can be identified by PRRs of microglia, activate the TLR/NF-κ B inflammatory pathway, and promote the release of pro-inflammatory factor cytokines and chemokines (119)

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39 These findings show that AUR/BSO and AUR/ERA cotreatment triggers typical parameters of intrinsic apoptosis, although cell death can proceed in a caspase-independent manner when caspases are blocked