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Glucose can be bound to hemoglobin through the N-terminal residue of the protein and also some side chain ε-amino groups of lysine residues, and the extent of glycation depends on the time and the level of glycemia

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If the apoptotic cells are not phagocytosed immediately, they undergo post-translational modification altering antigenicity that may provide a source of nuclear antigens to drive the autoantibody response in SLE [4]