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NF-κB can be activated by two different pathways: the “canonical” and the “alternative,” keeping in common the presence of different inhibitors from the IκB family (transcriptional repressors that halt NF-κB in the cytosol), and the IκB kinase family (IKK

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At this point, the answer is more restrained: Epitalon is biologically interesting, mechanistically plausible in some settings, and supported by a limited but intriguing body of preclinical and early translational literature

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