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Research indicates that ether-bonded glycerolipids increase cellular sensitivity to ferroptosis, whereas ether lipids are crucial for the formation of the extracellular meshwork induced by sulfasalazine (104)
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In mouse histiocytomas, oligomycin treatment reduced cellular ATP levels and significantly inhibited apoptosis, and also oligomycin inhibited caspase-1 and caspase-3 activities and loss of mitochondrial membrane potential, suggesting that the inhibition of apoptosis by oligomycin may be due to inhibition of ATP production, caspase activity and inhibition of mitochondrial depolarization (Singh and Khar, 2005)
