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Our results point to a novel metabolic vulnerability of pancreatic CSCs that should be exploited for the design of new therapeutic strategies aimed at the elimination of this highly aggressive subpopulation of cancer cells

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This is further supported by the fact that mice deficient in the Gclm gene are viable but have markedly reduced tissue GSH levels (reduced by about 85 to 90%) (Yang et al., 2002)

glutamic acid and is naturally found in every cell in the body and provides protection from free radicals