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Research has examined effects involving: Extracellular matrix — collagen, proteoglycans, glycosaminoglycans, and matrix-remodeling enzymes Fibroblasts — cells responsible for producing and organizing many extracellular-matrix components Metalloproteinases — enzymes involved in degradation and remodeling of extracellular-matrix proteins Vascular biology — endothelial cells and angiogenic signaling Gene expression — experimental changes in expression of numerous genes following GHK exposure Inflammatory signaling — inflammatory mediators and oxidative processes This broad range of observations makes GHK-Cu an interesting model for studying cellular remodeling rather than a single isolated pathway