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Lidocaine is extensively metabolized in the liver, so drugs that decrease hepatic blood flow (e.g., propranolol) or inhibit CYP1A2/CYP3A4 (e.g., fluvoxamine, ketoconazole, clarithromycin) may elevate systemic concentrations and increase the risk of central nervous system or cardiovascular toxicity
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In contrast, both CJC-1295 and tesamorelin function upstream by engaging the GHRH receptor, leading to increased growth hormone release and subsequent activation of the IGF-1 pathway