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Glucose metabolism is accelerated by exposure to t-butylhydroperoxide during NADH consumption in human erythrocytes

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This is in agreement with the observations that mice in which either SelP (95) or its receptor, ApoER2 (96), were knocked out, displayed ataxia and seizures (97,98) and that those in which tRNA Sec was deleted from neurons had neurodevelopmental and degenerative phenotype in the cerebellum, hippocampus, and cerebral cortex (99)